Toll样受体信号通路中p38 MAP激酶活化的机制研究
基本信息
结项摘要
微生物等病原感染引发的炎症性疾病是导致人类死亡的主要因素。TLRs（toll-like receptors）是新近发现的一类识别各种病原微生物的受体. TLRs激发下游的p38、JNK（Jun N-terminal kinase）、ERK（extracellular signal-regulated protein kinase）、NF-κB（nuclear factor-κB）等，启动细胞因子的
项目摘要
项目成果
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数据更新时间：2023-05-31
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戈宝学的其他基金
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